1. Krumholz HM and others. Lack of association between cholesterol and coronary heart disease mortality and morbidity and all-cause mortality in persons older than 70 years. Journal of the American Medical Association 272, 1335-1340, 1990.
2. Ravnskov U. High cholesterol may protect against infections and atherosclerosis. Quarterly Journal of Medicine 96, 927-934, 2003.
3. Jacobs D and others. Report of the conference on low blood cholesterol: Mortality associations. Circulation 86, 1046–1060, 1992.
4. Iribarren C and others. Serum total cholesterol and risk of hospitalization, and death from respiratory disease. International Journal of Epidemiology 26, 1191–1202, 1997.
5. Iribarren C and others. Cohort study of serum total cholesterol and in-hospital incidence of infectious diseases. Epidemiology and Infection 121, 335–347, 1998.
6. Claxton AJ and others. Association between serum total cholesterol and HIV infection in a high-risk cohort of young men. Journal of acquired immune deficiency syndromes and human retrovirology 17, 51–57, 1998.
7. Neaton JD, Wentworth DN. Low serum cholesterol and risk of death from AIDS. AIDS 11, 929–930, 1997.
8. Rauchhaus M and others. Plasma cytokine parameters and mortality in patients with chronic heart failure. Circulation 102, 3060-3067, 2000.
9. Niebauer J and others. Endotoxin and immune activation in chronic heart failure. Lancet 353, 1838-1842, 1999.
10. Vredevoe DL and others. Skin test anergy in advanced heart failure secondary to either ischemic or idiopathic dilated cardiomyopathy. American Journal of Cardiology 82, 323-328, 1998.
11. Rauchhaus M, Coats AJ, Anker SD. The endotoxin-lipoprotein hypothesis. Lancet 356, 930–933, 2000.
12. Rauchhaus M and others. The relationship between cholesterol and survival in patients with chronic heart failure. Journal of the American College of Cardiology 42, 1933-1940, 2003.
13. Horwich TB and others. Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure. Journal of Cardiac Failure 8, 216-224, 2002.
14. Elias ER and others. Clinical effects of cholesterol supplementation in six patients with the Smith-Lemli-Opitz syndrome (SLOS). American Journal of Medical Genetics 68, 305–310, 1997.
15. Bhakdi S and others. Binding and partial inactivation of Staphylococcus aureus a-toxin by human plasma low density lipoprotein. Journal of Biological Chemistry 258, 5899-5904, 1983.
16. Flegel WA and others. Inhibition of endotoxin-induced activation of human monocytes by human lipoproteins. Infection and Immunity 57, 2237-2245, 1989.
17. Weinstock CW and others. Low density lipoproteins inhibit endotoxin activation of monocytes. Arteriosclerosis and Thrombosis 12, 341-347, 1992.
18. Muldoon MF and others. Immune system differences in men with hypo- or hypercholesterolemia. Clinical Immunology and Immunopathology 84, 145-149, 1997.
19. Feingold KR and others. Role for circulating lipoproteins in protection from endotoxin toxicity. Infection and Immunity 63, 2041-2046, 1995.
20. Netea MG and others. Low-density lipoprotein receptor-deficient mice are protected against lethal endotoxemia and severe gram-negative infections. Journal of Clinical Investigation 97, 1366-1372, 1996.
21. Harris HW, Gosnell JE, Kumwenda ZL. The lipemia of sepsis: triglyceride-rich lipoproteins as agents of innate immunity. Journal of Endotoxin Research 6, 421-430, 2001.
22. Netea MG and others. Hyperlipoproteinemia enhances susceptibility to acute disseminated Candida albicans infection in low-density-lipoprotein-receptor-deficient mice. Infection and Immunity 65, 2663-2667, 1997.
23. Ross R, Glomset JA. The pathogenesis of atherosclerosis. New England Journal of Medicine 295, 369-377, 1976.
24. Ross R. The pathogenesis of atherosclerosis and update. New England Journal of Medicine 314, 488-500, 1986.
25. Klotz O, Manning MF. Fatty streaks in the intima of arteries. Journal of Pathology and Bacteriology. 16, 211-220, 1911.
26. At least 200 reviews about the role of infections in atherosclerosis and cardiovascular disease have been published; here are a few of them: a) Grayston JT, Kuo CC, Campbell LA, Benditt EP. Chlamydia pneumoniae strain TWAR and atherosclerosis. European Heart Journal Suppl K, 66-71, 1993. b) Melnick JL, Adam E, Debakey ME. Cytomegalovirus and atherosclerosis. European Heart Journal Suppl K, 30-38, 1993. c) Nicholson AC, Hajjar DP. Herpesviruses in atherosclerosis and thrombosis. Etiologic agents or ubiquitous bystanders? Arteriosclerosis Thrombosis and Vascular Biology 18, 339-348, 1998. d) Ismail A, Khosravi H, Olson H. The role of infection in atherosclerosis and coronary artery disease. A new therapeutic target. Heart Disease 1, 233-240, 1999. e) Kuvin JT, Kimmelstiel MD. Infectious causes of atherosclerosis. f.) Kalayoglu MV, Libby P, Byrne GI. Chlamydia pneumonia as an emerging risk factor in cardiovascular disease. Journal of the American Medical Association 288, 2724-2731, 2002.
27. Grau AJ and others. Recent bacterial and viral infection is a risk factor for cerebrovascular ischemia. Neurology 50, 196-203, 1998.
28. Mattila KJ. Viral and bacterial infections in patients with acute myocardial infarction. Journal of Internal Medicine 225, 293-296, 1989.
29. The successful trials: a) Gurfinkel E. Lancet 350, 404-407, 1997. b) Gupta S and others. Circulation 96, 404-407, 1997. c) Muhlestein JB and others. Circulation 102, 1755-1760, 2000. d) Stone AFM and others. Circulation 106, 1219-1223, 2002. e) Wiesli P and others. Circulation 105, 2646-2652, 2002. f) Sander D and others. Circulation 106, 2428-2433, 2002.
30. The unsuccessful trials: a) Anderson JL and others. Circulation 99, 1540-1547, 1999. b) Leowattana W and others. Journal of the Medical Association of Thailand 84 (Suppl 3), S669-S675, 2001. c) Cercek B and others. Lancet 361, 809-813, 2003. d) O’Connor CM and others. Journal of the American Medical Association. 290, 1459-1466, 2003.
31. Gieffers J and others. Chlamydia pneumoniae infection in circulating human monocytes is refractory to antibiotic treatment. Circulation 104, 351-356, 2001
32. Gurfinkel EP and others. Circulation 105, 2143-2147, 2002.
About the author
Dr. Ravnskov is the author of The Cholesterol Myths and chairman of The International Network of Cholesterol Skeptics (thincs.org).
There is one risk factor that is known to be certain to cause death. It is such a strong risk factor that it has a 100 percent mortality rate. Thus I can guarantee that if we stop this risk factor, which would take no great research and cost nothing in monetary terms, within a century human deaths would be completely eliminated. This risk factor is called “Life.”
Barry Groves, second-opinions.com
Familial Hypercholesterolemia – Not as Risky as You May Think
Many doctors believe that most patients with familial hypercholesterolemia (FH) die from CHD at a young age. Obviously, they do not know the surprising finding of the Scientific Steering Committee at the Department of Public Health and Primary Care at Radcliffe Infirmary in Oxford, England. For several years, these researchers followed more than 500 FH patients between the ages of 20 and 74 and compared patient mortality during this period with that of the general population.
During a three- to four-year period, six of 214 FH patients below age 40 died from CHD. This may not seem particularly frightening but as it is rare to die from CHD before the age of 40, the risk for these FH patients was almost 100 times that of the general population.
During a four- to five-year period, eight of 237 FH patients between ages 40 and 59 died, which was five times more than the general population. But during a similar period of time, only one of 75 FH patients between the ages of 60 and 74 died from CHD, when the expected number was two.
If these results are typical for FH, you could say that between ages 20 and 59, about 3 percent of the patients die from CHD, and between ages 60 and 74, less than 2 percent die, in both cases during a period of 3-4 years. The authors stressed that the patients had been referred because of a personal or family history of premature vascular disease and therefore were at a particularly high risk for CHD. Most patients with FH in the general population are unrecognized and untreated. Had the patients studied been representative for all FH patients, their prognosis would probably have been even better.
This view was recently confirmed by Dr. Eric Sijbrands and his coworkers from various medical departments in Amsterdam and Leiden, Netherlands. Out of a large group they found three individuals with very high cholesterol. A genetic analysis confirmed the diagnosis of FH and by tracing their family members backward in time, they came up with a total of 412 individuals. The coronary and total mortality of these members were compared with the mortality of the general Dutch population.
The striking finding was that those who lived during the 19th and early 20th century had normal mortality and lived a normal life span. In fact, those living in the 19th century had a lower mortality than the general population. After 1915 the mortality rose to a maximum between 1935 and 1964, but even at the peak, mortality was less than twice as high as in the general population.
Again, very high cholesterol levels alone do not lead to a heart attack. In fact, high cholesterol may even be protective against other diseases. This was the conclusion of Dr. Sijbrands and his colleagues. As support they cited the fact that genetically modified mice with high cholesterol are protected against severe bacterial infections.
“Doctor, don’t be afraid because of my high cholesterol.” These were the words of a 36-year-old lawyer who visited me for the first time for a health examination. And indeed, his cholesterol was high, over 400 mg/dl.
“My father’s cholesterol was even higher,” he added. “But he lived happily until he died at age 79 from cancer. And his brother, who also had FH, died at age 83. None of them ever complained of any heart problems.” My “patient” is now 53, his brother is 56 and his cousin 61. All of them have extremely high cholesterol values, but none of them has any heart troubles, and none of them has ever taken cholesterol-lowering drugs.
So, if you happen to have FH, don’t be too anxious. Your chances of surviving are pretty good, even surviving to old age.
Scientific Steering Committee on behalf of the Simon Broome Register Group. Risk of fatal coronary heart disease in familial hypercholesterolaemia. British Medical Journal 303, 893-896, 1991; Sijbrands EJG and others. Mortality over two centuries in large pedigree with familial hypercholesterolaemia: family tree mortality study. British Medical Journal 322, 1019-1023, 2001.
From The Cholesterol Myths by Uffe Ravnvskov, MD, PhD, NewTrends Publishing, pp 64-65.
See also: Uffe Ravnskov, MD, PhD New cholesterol guidelines for converting healthy people into patients
Summary of latest data on antibiotic resistance in the European Union
Highlights on antibiotic resistance Antibiotic resistance is a major global public health problem and is, for a large part, driven by use of antibiotics. As a result, patients are suffering from infections caused by bacteria that are resistant to antibiotics, sometime to multiple antibiotics. Resistance in bacteria commonly responsible for infections such as Klebsiella pneumoniae and Escherichia coli has been increasing Europe wide for all Antibiotic classes under surveillance. Single and combined resistance to several antibiotics (multidrug resistance) are increasing in these bacteria. The percentage of carbapenem -Resistant lebsiella pneumonia is increasing in the European Union and these resistant bacteria are now spreading to several European countries . Transfer of patients across borders poses a clear risk for the transmission of carbapenem resistant bacteria, especially when patients are transferred from areas with high rates of such bacteria to healthcare facilities in another country or have received medical care abroad in areas with high rates of carbapenem resistant bacteria.Carbapenems are the major last line class of antibiotics to treat infections with multidrug resistant Gram negative bacteria such as K. pneumoniae and E. coli Resistance to carbapenems limits available options for treatment of infected patients to only few antibiotics which often are old antibiotics that were developed several decades ago and often have limitations and side effects . The occurrence of meticillin resistant Staphylococcus aureus (MRSA) shows a decrease in some European countries. However, one fourth of countries are still reporting that of all Staphylococcus aureus invasive infections, more than 25% are MRSA. Antibiotic resistance In the European Union The data presented in this section were collected by the European Antimicrobial Resistance Surveillance Network (EARS Net) which is coordinated by the European Centre for Disease Prevention and Control (ECDC). The maps presented in this summary show the occurrence of antibiotic resistance in selected bacteria causing invasive infections and are based on laboratory results reported by countries participating in EARS Net. 2 Klebsiella pneumoniae ( K. pneumoniae ) K. pneumoniae is one of the common causes of Gram negative urinary and respiratory tract infections. This micro organism can rapidly spread from the gastrointestinal tract of patients and via the hands of the hospital personnel to other patients, leading to nosocomial outbreaks. Importantly, patients with impaired immune defences are at higher risk to acquire these infections. The percentage of carbapenem resistant K. pneumoniae has increased dramatically in the European Union since 2009–In 2009, carbapenem resistance in K. pneumoniae was only established in Greece, although carbapenem resistance in invasive isolates of K. pneumoniae was also found in Italy and Cyprus [F2]. In 2010, an increasing trend of carbapenem –
resistant K. pneumonia e was observed for Austria, Cyprus, Hungary and Italy (Fig. 1). This increasing trend is a particularly worrying phenomenon since carbapenems are last line antibiotics for treatment of infections with multidrug Resistant Gram negative bacteria including those which produce an extended spectrum beta lactamase (ESBL). Treatment options for patients infected with carbapenem resistant K. pneumoniae or other carbapenem resistant bacteria are severely limited [F3]3 Figure 1 Klebsiella pneumoniae : percentage of invasive isolates resistant to carbapenems in 2009 (A) and in 2010 (B) (Data source: EARS Net) Percentage of carbapenem resistant K. pneumoniae in 2009 B:Percentage of carbapenem resistant K. pneumoniae in 2010
4 In addition to resistance to carbapenems, a high frequency of Multidrug resistant K. pneumoniae (resistance to third generation cephalosporins, fluoroquinolones and aminoglycosides ) is evident in Southern, Central and Eastern Europe (Fig.2). Figure 2: Kleb siella pneumoniae percentage of multidrug resistant K. pneumoniae (third generation cephalosporins, fluoroquinolones and aminoglycosides) in 2010 (Data source: EARS Net)
Escherichia coli ( E. coli ) E. coli is the most frequent cause of bacteraemia caused by Gram negative bacteria, as well as community and hospital – acquired urinary tract infections. It is also one of the most common foodborne pathogens worldwide. The occurrence of antibiotic resistance in E. coli continues to increase Europe wide for both multidrug resistance and for single antibiotics under surveillance. For some antibiotics ,the increase is evident even among countries already presenting relatively high levels of resistance. For fluoroquinolones, which are important antibiotics for treatment of E. coli infections, the resistance situation in Europe in 2010 is displayed in
6 Figure 3 Escherichia coli Percentage of invasive isolates with resistance to fluoroquinolones in 2009 and 2010 ( Data source EARS Net)
A:Percentage of E. coli resistant to fluoroquinolones in 2009 B
Percentage of E. coli resistant to fluoroquinolones in 2010 7 Meticillin –
resistant Staphylococcus aureus (MRSA ) MRSA is the most important cause of antibiotic – resistant healthcare -associated infections worldwide. Infections with MRSA may result in prolonged hospital stay and increased mortality rates[F4]. In 2010, seven countries ( Austria, Cyprus, Estonia, France, Greece, Ireland and the UK ) reported decreasing trends for MRSA. This brings hope that national efforts on infection control and containment of resistance may in some cases slow down the development of resistance. Nevertheless , MRSA remains a public health priority, since significantly increasing trend of MRSA was observed in four countries (Italy, Hungary, Germany and Slovenia ) and the proportion of MRSA remains above 25% in more than one fourth of countries (Fig. 4). 8
Figure 4 Staphylococcus aureus Percentage of invasive isolates resistant to meticillin (MRSA) in 2009 A) and in 2010 (B) (Data source EARS Net)
A: Percentage of MRSA in 2009
B: Percentage of MRSA in 2010
Recipe for Insulin Regulating—take Fenugreek and Ginger Powder—equal portions and either make a tea with this or fuse with Honey ( Unpasteurized) –when fused Utilize ¼ tsp of this twice a day or as needed—you will find this will reduce hunger as a direct result of insulin regulating and may find as well the benefits of the components of these herbs—Or brew a tea with this and drink throughout the day – may as well see GH response from this as well Hormonal Levels for regenerating the Body and muscle integrity –potential Testosterone Regulating as well—Estrogen Blocking—Immune Support
Flavonoids Flavone (apigenin, luteolin) glycosides including
orientin and vitexin, quercetin (flavonol).
Saponins 0.6–1.7%. Glycosides yielding steroidal sapogenins
diosgenin and yamogenin (major), with tigogenin, neotigogenin,
gitogenin, neogitogenin, smilagenin, sarsasapogenin, yuccagenin;(
1) fenugreekine, a sapogenin-peptide ester involving diosgenin
and yamogenin;(2) trigofoenosides A–G (furostanol
Other constituents Coumarin,(7) lipids (5–8%),(8) mucilaginous
fibre (50%),(8) vitamins (including nicotinic acid) and minerals
Proteins and amino acids Protein (23–25%) containing high
quantities of lysine and tryptophan. Free amino acids include 4-
hydroxyisoleucine (0.09%), histidine, lysine and arginine
Oleo-resin Gingerol homologues (major, about 33%) including
derivatives with a methyl side-chain,(4) shogaol homologues
(dehydration products of gingerols), zingerone (degradation
product of gingerols), 1-dehydrogingerdione,(5) 6-gingesulfonic
acid(3) and volatile oils.
Volatile oils 1–3%. Complex, predominately hydrocarbons. b-
Bisabolene and zingiberene (major); other sesquiterpenes include
zingiberol, zingiberenol, ar-curcumene, b-sesquiphellandrene, bsesquiphellandrol
(cis and trans); numerous monoterpene hydrocarbons,
alcohols and aldehydes (e.g. phellandrene, camphene,
geraniol, neral, linalool, d-nerol).
Proteins and amino acids
Amino acids (e.g. arginine, aspartic acid,
cysteine, glycine, isoleucine, leucine, serine, threonine and valine),
protein (about 9%), resins, diterpenes (galanolactone),(6) vitamins
(especially nicotinic acid (niacin) and vitamin A), minerals.(2)
The material contains not less than 4.5% of alcohol (90%)-
soluble extractive and not less than 10% of water-soluble
Eggs Prevent Heart Disease and Cancer
New Evidence That Egg White Protein May Help High Blood Pressure
Apr. 9, 2013 — NEW ORLEANS, April 9, 2013 — Scientists reported new evidence today that a component of egg whites –– already popular as a substitute for whole eggs among health-conscious consumers concerned about cholesterol in the yolk –– may have another beneficial effect in reducing blood pressure. Their study was part of the 245th National Meeting & Exposition of the American Chemical Society (ACS), the world’s largest scientific society, which continues here through Thursday.—“Our research suggests that there may be another reason to call it ‘the incredible, edible egg,’” said study leader Zhipeng Yu, Ph.D., of Jilin University. “We have evidence from the laboratory that a substance in egg white –– it’s a peptide, one of the building blocks of proteins –– reduces blood pressure about as much as a low dose of Captopril, a high-blood-pressure drug.”—Yu and colleagues, who are with Clemson University, used a peptide called RVPSL. Scientists previously discovered that the substance, like the family of medications that includes Captopril, Vasotec and Monopril, was an angiotensin-converting-enzyme (ACE) inhibitor. It has a powerful ability to inhibit or block the action of ACE, a substance produced in the body that raises blood pressure.—They set out to further document RVPSL’s effects, using laboratory rats that develop high blood pressure and are stand-ins for humans in such early research on hypertension. The results of feeding the substance were positive, showing that RVPSL did not have apparent toxic effects and lowered blood pressure by amounts comparable to low doses of Captopril.—“Our results support and enhance previous findings on this topic,” Yu said. “They were promising enough to move ahead with further research on the effects of the egg white peptide on human health.”—Yu noted that the research was done with a version of the peptide that was heated to almost 200 degrees Fahrenheit during preparation — less than the temperatures typically used to cook eggs. He cited evidence from other research, however, that egg whites may retain their beneficial effects on blood pressure after cooking.–One, for instance, published in the ACS’ Journal of Agricultural and Food Chemistry, showed that fried egg protein, cooked at high temperatures, actually showed greater ability to reduce blood pressure than eggs boiled at 212 degrees F.–Yu believes that egg white peptides, either in eggs or as a supplement, could become useful as an adjunct to high-blood-pressure medication. For now, he said people with high blood pressure should consult their health care provider before making any changes. –And he noted that findings about egg white and high blood pressure add to the emerging nutritional image of eggs. Once regarded as a food to avoid in a healthy diet, studies in recent years have concluded that many people can eat eggs without raising their blood cholesterol levels, benefiting from an inexpensive food low in calories and rich in protein, vitamins and other nutrients.—The American Chemical Society is a nonprofit organization chartered by the U.S. Congress. With more than 163,000 members, ACS is the world’s largest scientific society and a global leader in providing access to chemistry-related research through its multiple databases, peer-reviewed journals and scientific conferences. Its main offices are in Washington, D.C., and Columbus, Ohio.
Eggs’ Antioxidant Properties May Help Prevent Heart Disease and Cancer, Study
July 6, 2011 — One of nature’s most perfect foods may be even better for us than previously thought.—While eggs are well known to be an excellent source of proteins, lipids, vitamins and minerals, researchers at the University of Alberta recently discovered they also contain antioxidant properties, which helps in the prevention of cardiovascular disease and cancer. –Jianping Wu, Andreas Schieber and graduate students Chamila Nimalaratne and Daise Lopes-Lutz of the U of A Department of Agricultural Food and Nutritional Science examined egg yolks produced by hens fed typical diets of either primarily wheat or corn. They found the yolks contained two amino acids, tryptophan and tyrosine, which have high antioxidant properties. –After analyzing the properties, the researchers determined that two egg yolks in their raw state have almost twice as many antioxidant properties as an apple and about the same as half a serving (25 grams) of cranberries.—However, when the eggs were fried or boiled, antioxidant properties were reduced by about half, and a little more than half if the eggs were cooked in a microwave.[F5] -“It’s a big reduction but it still leaves eggs equal to apples in their antioxidant value,” said Wu.–The findings were published in the peer-reviewed journal Food Chemistry. –The discovery of these two amino acids, while important, may only signify the beginning of finding antioxidant properties in egg yolks, said Wu, an associate professor of agricultural, food and nutritional science.–“Ultimately, we’re trying to map antioxidants in egg yolks so we have to look at all of the properties in the yolks that could contain antioxidants, as well as how the eggs are ingested,” said Wu, adding that he and his team will examine the other type of antioxidant already known to be in eggs, carotenoids, the yellow pigment in egg yolk, as well as peptides. ( see list below )—In previous research, Wu found that egg proteins were converted by enzymes in the stomach and small intestines and produced peptides that act the same way as ACE inhibitors, prescriptions drugs that are used to lower high blood pressure.–That finding defied common wisdom and contradicted the public perception that eggs increased high blood pressure because of their high cholesterol content. Additional research by Wu suggests the peptides can be formulated to help prevent and treat hypertension.—Wu is convinced the peptides also have some antioxidant properties, which leads him to suggest that when he completes the next step in his research, the result will likely be that eggs have more antioxidant properties than we currently know.
Other Antioxidants as well as Fats crucial for regenerating
Eggs Contain these Substances note
(mg of Substance per 100 grams)
Alkaloids: Betaine 0.53
Amino Acids: Methionine Neurine *
Carotenoids: Xanthophylls: * Lutein Zeaxanthin
Lipids: 11,200 Arachidonic Acid Phosphatidylcholine 1,875
Cholesterol 371 -548 Docosahexaenoic Acid ( DHA)
Linoleic Acid Alpha-Linolenic Acid ( Omega 3)
Stearic Acid Sphingomyelin 10.74
Minerals: Calcium 52 Chromium* 16 mcg
Zinc* 1.5 Sodium 140
Potassium 140 Iron 2
Proteins: ** Avidin # Ovalbumin *
Ovomucoid * Ovoglobulin *
Conalbumin * Vitellin*
Vitellenin * Ovomucin #
Levitin Albumin #
Vitamins: Biotin* 25 mcg Choline * 251
Vitamin A 140 mcg Vitamin B5
Vitamin B1 0.09 Vitamin B2 0.47
Vitamin D 50 IU Vitamin E 1.6
Folic Acid (Yolks only) 25 mcg Vitamin B12 1.7 mcg
Vitamin K * Vitamin B3 3.7
* Egg Yolks only
# Egg Whites only
** The biological value of the Proteins in Egg Whites is 88
– The biological value of the Proteins in whole Eggs is 100.
[F1]And then you wonder why kids are spun and grown ups cannot think ir remember anything
[F2]The Mediterranean Region—so this would be what is recorded —the concern would be what is not recorded
[F3]In cases like this then you will utilize iodine –turpentine—essential oils –and minerals such as zinc –copper—salt –and a combinations of salt with other elements to insure penetration and removal—what is going on is that these pathogens are forming a biofilm polymer that will layer inside a body developing a protective shield –to eliminate a immune response while it feeds on the host utilizing the bodies own resources to further cascade the system and take over—a rewriting of the genetic code—which may be tied to direct infiltration through vaccines and Genetically modified foods which are loaded with viral –fungal and bacterial elements not normally seen in the balanced biology of botany—even the soil in which they are grown are being shown to have issues and cause a furthering of genetic distortion in the plants—further exasberating there damage internally—further causing the infections to breakdown the host
[F4]It is completely curable with either essential oils –iodine or turpentine—even botanicals in combination with minerals will remove this
[F5]Never use a Microwave–EVER
Show of the Month April 27 2013
Antibiotic Overload In feed
Caffeine and Exercise May Be Protective Against Skin Cancer Caused by Sun Exposure
Caffeine Shown As Effective At Reducing Exercise-Induced Asthma Symptoms As An Albuterol Inhaler
Immune-modifying and antimicrobial effects of Eucalyptus oil and simple inhalation devices.
Antibiotic Overload In feed
Did you know that each year more antibiotics are fed to food-animals in North Carolina than are given to all Americans? Thanks to this kind of misuse, antibiotic-resistant diseases now kill more Americans than HIV/AIDS.—On March 9, 2011, Rep. Slaughter re-introduced H.R. 965, The Preservation of Antibiotics for Medical Treatment Act (PAMTA), which is designed to end the routine use of antibiotics on healthy animals and curb the growing threat of superbugs.—PAMTA would preserve the effectiveness of medically important antibiotics by phasing out the use of these drugs in healthy food-producing animals, while allowing their use for treatment of sick animals. The legislation also requires the Food and Drug Administration (FDA) to apply the same tough standards to new applications for approval of animal antibiotics.
On February 23, 2011, Slaughter confirmed with the FDA an alarming statistic: 80 percent of all antibiotics used in the United States are used not on humans but on food-animals, most of which are perfectly healthy.—This kind of habitual use of antibiotics has been conclusively linked to the growing risk of antimicrobial-resistant infections in humans. A National Academy of Sciences report stated that, “a decrease in antimicrobial use in human medicine alone will have little effect on the current situation. Substantial efforts must be made to decrease inappropriate overuse in animals and agriculture as well.”—When our limited supply of antibiotics is used indiscriminately and without care, there are public health consequences. It is time to put a stop to big agribusinesses doling out pharmaceuticals to healthy animals just because it is better for their bottom line. Antibiotic use in food-animals must be limited to prevent the inadvertent creation of superbugs that are too powerful for our own medicine. In addition to introducing legislation, Slaughter has called upon the FDA and the United States Department of Agriculture (USDA) to take action to improve regulatory oversight[F1], surveillance, and monitoring of food-animal production and antibiotic resistance. Last year, the Governmental Accounting Agency (GAO) released a report that Rep. Slaughter requested in 2009, which found that federal agencies have made limited progress in combating the growing threat of antibiotic resistance.—On March 11, 2011, Slaughter led a letter to Secretary Sebelius at the Department of Health and Human Services to urge the formation of a national plan to combat antibiotic resistance. Since that letter was written, a national plan to combat antibiotic resistance has been issued.
Slaughter’s work continues to raise awareness of this important issue. On December 13, 2011, she hosted a briefing in which farmers and successful businesses extolled the benefits of tapping into the growing domestic and international demand for antibiotic-free meat.—Slaughter was joined by an impressive panel, including Steve Ells, CEO of Chipotle Mexican Grill, one of the nation’s fastest growing restaurant companies, Stephen McDonnell, CEO of award-winning Applegate Farms, and Paul Willis, President of Niman Ranch, a network of over 650 independent sustainable farms. To learn more about the briefing, click here.—–As businesses such as Applegate Farms, Niman Ranch, the Ozark Mountain Pork Cooperative, and Chipotle Mexican Grill, continue to pioneer affordable, antibiotic-free meat, Slaughter continues to push the federal government to address this looming public health threat.—-Earlier this year, Slaughter sent letters to over 60 fast food companies, producers, processors, and grocery store chains asking them to disclose their policies on antibiotic use in meat and poultry production. Very simply, consumers have a right to know what is in their food. The U.S. is facing a growing public health crisis in the form of antibiotic-resistant bacteria, and information about how companies may or may not be contributing to the problem should be available to consumers. To view Slaughter’s letter, click here.
Edible Parts: Inner bark.
Edible Uses: Condiment.
Inner bark – dried and ground into a powder and used in making bread[2, 66, 105, 177]. It is often mixed with oatmeal. A famine food, it is only used when all else fails. A vanillin flavouring is obtained as a by-product of other resins that are released from the pulpwood.
Antirheumatic; Antiseptic; Aromatherapy; Bach; Balsamic; Diuretic; Expectorant.
Utilizing Scent-or Vapour
The resinous materials
Scot’s pine has quite a wide range of medicinal uses, being valued especially for its antiseptic action and beneficial effect upon the respiratory system. It should not be used by people who are prone to allergic skin reactions whilst the essential oil should not be used internally unless under professional supervision. The turpentine obtained from the resin is antirheumatic, antiseptic, balsamic, diuretic, expectorant, rubefacient and vermifuge[4, 13, 46]. It is a valuable remedy in the treatment of kidney, bladder and rheumatic affections, and also in diseases of the mucous membranes and the treatment of respiratory complaints. Externally it is used in the form of liniment plasters and inhalers. The leaves and young shoots are antiseptic, diuretic and expectorant. They are harvested in the spring and dried for later use. They are used internally for their mildly antiseptic effect within the chest and are also used to treat rheumatism and arthritis. They can be added to the bath water for treating fatigue, nervous exhaustion, sleeplessness, skin irritations. They can also be used as an inhalant in the treatment of various chest complaints. The essential oil from the leaves is used in the treatment of asthma, bronchitis and other respiratory infections, and also for digestive disorders such as wind. An essential oil obtained from the seed has diuretic and respiratory-stimulant properties. The seeds are used in the treatment of bronchitis, tuberculosis and bladder infections. A decoction of the seeds can be applied externally to help suppress excessive vaginal discharge. The plant is used in Bach flower remedies – the keywords for prescribing it are ‘Self-reproach’, ‘Guilt feelings’ and ‘Despondency'. The essential oil is used in aromatherapy. Its keyword is ‘Invigorating'.
Dye; Essential; Fibre; Fuel; Herbicide; Lighting; Packing; Resin; Shelterbelt; Wood.
A tan or green dye is obtained from the needles. The needles contain a substance called terpene, this is released when rain washes over the needles and it has a negative effect on the germination of some plants, including wheat. A reddish yellow dye is obtained from the cones. This tree yields resin and turpentine[64, 66, 100, 171]. Oleo-resins are present in the tissues of all species of pines, but these are often not present in sufficient quantity to make their extraction economically worthwhile. The resins are obtained by tapping the trunk, or by destructive distillation of the wood[4, 64]. In general, trees from warmer areas of distribution give the higher yields. Turpentine consists of an average of 20% of the oleo-resin and is separated by distillation[4, 64]. Turpentine has a wide range of uses including as a solvent for waxes etc, for making varnish, medicinal etc. Rosin is the substance left after turpentine is removed. This is used by violinists on their bows and also in making sealing wax, varnish etc. Pitch can also be obtained from the resin and is used for waterproofing, as a wood preservative etc. An essential oil obtained from the leaves is used in perfumery and medicinally[46, 61]. A fibre from the inner bark is used to make ropes. The roots are very resinous and burn well. They can be used as a candle substitute. The leaves are used as a packing material. The fibrous material is stripped out of the leaves and is used to fill pillows, cushions and as a packing material. Trees are very wind resistant and quite fast growing. They can be planted as a shelterbelt, succeeding in maritime exposure[75, 200]. Wood – light, soft, not strong, elastic, durable, rich in resin. Used in construction, furniture, paper manufacture etc.[13, 46, 100]. A good fuel but it is somewhat smokey[6, 66, 115].
Edible Parts: Inner bark.
Edible Uses: Condiment; Gum; Tea.
Inner bark – cooked. It is usually dried, ground into a powder and then used as a thickening in soups etc or mixed with cereals when making bread[105, 177]. Fir bark is a delight to chew in winter or early spring, slightly mucilaginous and sweetish, better raw than cooked. Another report says that it is an emergency food and is only used when all else fails. An aromatic resinous pitch is found in blisters in the bark. When eaten raw it is delicious and chewy[101, 183]. Another report says that the balsam or pitch, in extreme emergency, forms a highly concentrated, though disagreeable, food. An oleoresin from the pitch is used as a flavouring in sweets, baked goods, ice cream and drinks. Tips of young shoots are used as a tea substitute[177, 183].
Plants For A Future can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.
Analgesic; Antiscorbutic; Antiseptic; Diuretic; Poultice; Stimulant; Tonic; VD.
Cleaner of Wounds
Patch to draw out infections
Energizing or increases Alertness or flow
And Vitality Support
The resin obtained from the balsam fir (see ‘Uses notes’ below) has been used throughout the world and is a very effective antiseptic and healing agent. It is used as a healing and analgesic protective covering for burns, bruises, wounds and sores[213, 222, 226]. It is also used to treat sore nipples and is said to be one of the best curatives for a sore throat. The buds, resin, and/or sap are used in folk remedies for treating cancers, corns, and warts. The resin is also antiscorbutic, diaphoretic, diuretic, stimulant and tonic[4, 171, 222]. It is used internally in propriety mixtures to treat coughs and diarrhoea, though taken in excess it is purgative. A warm liquid of the gummy sap was drunk as a treatment for gonorrhoea. A tea made from the leaves is antiscorbutic[4, 171]. It is used in the treatment of coughs, colds and fevers. The leaves and young shoots are best harvested in the spring and dried for later use. This plant was widely used medicinally by various North American Indian tribes. The resin was used as an antiseptic healing agent applied externally to wounds, sores, bites etc., it was used as an inhalant to treat headaches and was also taken internally to treat colds, sore throats and various other complaints.
Adhesive; Fibre; Gum; Kindling; Microscope; Repellent; Resin; Stuffing; Waterproofing; Wood.
The balsamic resin ‘Balm of Gilead'[11, 46] or ‘Canada Balsam’ according to other reports[64, 226, 238] is obtained during July and August from blisters in the bark or by cutting pockets in the wood. Another report says that it is a turpentine. The term Canada Balsam is a misnomer because balsams are supposed to contain benzoic and cinnamic acids, both absent from the Canada oleoresin. Turpentine is also a misnomer, implying that the oleoresin is entirely steam volatile. Actually it contains 70 – 80% resin, only 16 – 20% volatile oil. Canada Balsam yields 15 – 25% volatile oil, the resin being used for caulking and incense. It is used medicinally and in dentistry, also in the manufacture of glues, candles and as a cement for microscopes and slides – it has a high refractive index resembling that of glass[11, 46, 64, 82, 222, 226, 238]. The pitch has also been used as a waterproofing material for the seams of canoes. The average yield is about 8 – 10 oz per tree. The resin is also a fixative in soaps and perfumery[171, 238]. “Turpentine” is usually collected during July-August by breaking the turpentine blisters into small metal cans with sharp-pointed lids. Trees are then allowed to recuperate for 1 – 2 years before being harvested again. The leaves and young branches are used as a stuffing material for pillows etc – they impart a pleasant scent[46, 61, 257] and also repel moths. The leaves contain an average of 0.65% essential oil, though it can go up to 1.4% or even higher. One analysis of the essential oils reports 14.6% bornyl acetate, 36.1% b-pinene, 11.1% 3-carene, 11.1% limonene, 6.8% camphene, and 8.4% a-pinene. To harvest the oil, it would appear that the branches should be snipped off younger trees in early spring. Fifteen year old trees yield 70% more leaf oil than 110-year-old trees; oil yields are highest in January – March and September, they are lowest from April to August. A thread can be made from the roots. Wood – light, soft, coarse grained, not strong, not very durable. Weighs 24lb per cubic foot. Used mainly for pulp, it is not used much for lumber except in the manufacture of crates etc[46, 82, 226, 229]. The wood is commercially valuable for timber even though it is relatively soft, weak, and perishable. Balsam fir is used in the US for timber and plywood, and is the mainstay of the pulp wood industry in the Northeast. The wood, which is rich in pitch, burns well and can be used as a kindling
Edible Uses: Condiment.
An essential oil from the fresh or dried leaves is used as a flavouring in sweets, baked goods, ice cream etc[177, 183].
Plants For A Future can not take any responsibility for any adverse effects from the use of plants. Always seek advice from a professional before using a plant medicinally.
Antibacterial; Antiperiodic; Antirheumatic; Antiseptic; Antispasmodic; Appetizer; Aromatherapy; Aromatic; Deodorant; Expectorant; Febrifuge;
Stops the flow of menses
Cleaner of Wounds
Stops spasms of muscles and nerves
Desire for food
Remedial use in aromatherapies
Scents Utilize for therapies
Free’s lungs from mucous build up
Sugar regulator for low sugar
Eucalyptus leaves are a traditional Aboriginal herbal remedy. The essential oil found in the leaves is a powerful antiseptic and is used all over the world for relieving coughs and colds, sore throats and other infections. The essential oil is a common ingredient in many over-the-counter cold remedies. The adult leaves, without their petioles, are antiperiodic, antiseptic, aromatic, deodorant, expectorant, febrifuge, hypoglycaemic and stimulant[4, 7, 21, 46]. The leaves, and the essential oil they contain, are antiseptic, antispasmodic, expectorant, febrifuge and stimulant. Extracts of the leaves have antibacterial activity. The essential oil obtained from various species of eucalyptus is a very powerful antiseptic, especially when it is old, because ozone is formed in it on exposure to air. It has a decided disinfectant action, destroying the lower forms of life. The oil can be used externally, applied to cuts, skin infections etc, it can also be inhaled for treating blocked nasal passages, it can be gargled for sore throat and can also be taken internally for a wide range of complaints. Some caution is advised, however, because like all essential oils, it can have a deleterious effect on the body in larger doses. The oil from this species has a somewhat disagreeable odour and so it is no longer used so frequently for medicinal purposes, other members of the genus being used instead. An oleo- resin is exuded from the tree. It can also be obtained from the tree by making incisions in the trunk[4, 152]. This resin contains tannin and is powerfully astringent, it is used internally in the treatment of diarrhoea and bladder inflammation[4, 152, 238], externally it is applied to cuts etc[4, 152]. The essential oil is used in aromatherapy. Its keyword is ‘Respiratory system'.
Cleanser; Deodorant; Dye; Essential; Fuel; Repellent; Wood.
The leaves and the essential oil in them are used as an insect repellent[14, 152, 174, 240]. The trees can also be planted in wet areas where mosquitoes abound. The ground will be dried out by the trees, making it unsuitable for the mosquitoes to breed. A decoction of the leaves is used for repelling insects and vermin. Africans use finely powdered bark as an insect dust. An essential oil is obtained from the leaves[46, 61, 156]. It is used in perfumery and in medicines. The yield is about 0.9% by steam distillation. The essential oil is also in spot removers for cleaning off oil and grease. Yields of 40 to 45 kilos of oil per hectare have been reported. A yellow/brown dye is obtained from the young leaves. It does not require a mordant. Grey and green dyes are obtained from the young shoots. A dark green dye is obtained from the young bark. Wood – heavy[46, 61], (or light according to another report), durable, fire resistant. An important timber species, it is used for various purposes such as carpentry, construction, fences, piles, platforms, plywood, poles, sheds, tool handles and veneer[238, 269]. The oil-rich wood is resistant to termites. This is one of the best eucalypts for pulp production for making paper[152, 269].
Caffeine and Exercise May Be Protective Against Skin Cancer Caused by Sun Exposure– Study Suggests
Apr. 3, 2012 — The combined effects of exercise plus caffeine consumption may be able to ward off skin cancer and also prevent inflammation related to other obesity-linked cancers.—“We found that this combination treatment can decrease sunlight-caused skin cancer formation in a mouse model,” said Yao-Ping Lu, Ph.D., associate research professor of chemical biology and director of skin cancer prevention at the Rutgers Ernest Mario School of Pharmacy in Piscataway, N.J. He presented these findings at the AACR Annual Meeting 2012, held in Chicago March 31 — April 4.–“I believe we may extrapolate these findings to humans and anticipate that we would benefit from these combination treatments as well,” Lu added.—The researchers evaluated the effects of caffeine and exercise on mice at high risk for developing skin cancer. Results showed that mice that took a dose of caffeine and exercised with a running wheel experienced 62 percent fewer skin tumors. The volume of tumors also decreased by 85 percent compared with the mice that did not consume caffeine or exercise.—-Positive effects were found with either caffeine or exercise alone, but to a lesser extent. Researchers observed a 27 percent reduction in tumors in caffeine-only mice and a 61 percent reduction in tumor size. In the exercise-only mice, researchers found that tumor activity decreased by 35 percent and tumor volume decreased by 70 percent.—The researchers also found that exercise and caffeine reduced weight and inflammation. They fed mice a high-fat diet of omega-6 fatty acid-rich foods and measured the volume of the parametrial fat pad (the largest fat pad in a mouse) after two weeks of exercise and/or caffeine treatment.—-Mice that had caffeine and exercised had a fat pad weight decrease of 63 percent. Caffeine-only mice had a 30 percent decrease, and exercise-only mice had a 56 percent decrease. Development and size of cancer decreased as well. The link, Lu believes, is inflammation, which dropped as much as 92 percent in mice that exercised and consumed caffeine.—This research was funded by the National Institutes of Health.- Story Source-The above story is reprinted from materials provided by American Association for Cancer Research (AACR).
Caffeine Shown As Effective At Reducing Exercise-Induced Asthma Symptoms As An Albuterol Inhaler
June 1, 2009 — An Indiana University study found that the ingestion of caffeine within an hour of exercise can reduce the symptoms of exercise induced asthma (EIA).
A large dose — 9 milligrams of caffeine per kilogram of body weight [F2]– was as effective as the use of an albuterol inhaler, which is commonly used to treat or prevent exercise-induced asthma. Smaller amounts of caffeine — for example, 3 and 6 milligrams of caffeine per kilogram of body weight — also reduced the wheezing, coughing and other symptoms of EIA.—Timothy Mickleborough, an associate professor in the Department of Kinesiology and co-investigator of the study, said no additional benefit was found when caffeine was combined with an albuterol inhaler.—Mickleborough and his research colleagues have been investigating the efficacy of a number of nutritional factors, and his research to date has shown that a diet high in fish oil and antioxidants and low in salt has the potential to reduce the severity of EIA and perhaps reduce the reliance on pharmacotherapy. This is especially important since prolonged use of daily medications can result in reduced effectiveness, and there is growing concern about the potential side effects of inhaled corticosteroid use.–*Background: The caffeine study involved 10 asthmatic subjects who also had EIA, in a randomized, double-blind double-dummy crossover study. They ingested 3, 6, or 9 milligrams of caffeine per kilogram of body weight or a placebo an hour before running on a treadmill. Pulmonary function tests were conducted 15 minutes before the a eucapnic voluntary hyperpnea challenge (a surrogate for an exercise challenge) and then again 1, 5, 10, 15 and 30 minutes afterward.–For someone weighing 150 pounds, 3 to 9 milligrams of caffeine per kilogram of body weight equals around 205 to 610 milligrams of caffeine. Earlier research has found that caffeine can reduce the symptoms of EIA. This study extends this earlier work and is the first to examine any synergistic effect of caffeine use along with an albuterol inhaler.—The study, “Comparative and Synergistic Effects of Caffeine and Albuterol on The Severity of Exercise-Induced Bronchoconstriction,” was presented at the American College of Sports Medicine conference during the Respiratory Session on May 29. Co-authors include lead author Timothy A. VanHaitsma, now at the University of Utah; Martin R. Lindley, Loughborough University, United Kingdom; and David Koceja and Joel Stager, IU’s Department of Kinesiology
Immune-modifying and antimicrobial effects of Eucalyptus oil and simple inhalation devices.
Sadlon AE, Lamson DW.
Eucalyptus oil (EO) and its major component, 1,8-cineole, have antimicrobial effects against many bacteria, including Mycobacterium tuberculosis and methicillin-resistant Staphylococcus aureus (MRSA), viruses, and fungi (including Candida). Surprisingly for an antimicrobial substance, there are also immune-stimulatory, anti-inflammatory, antioxidant, analgesic, and spasmolytic effects. Of the white blood cells, monocytes and macrophages are most affected, especially with increased phagocytic activity. Application by either vapor inhalation or oral route provides benefit for both purulent and non-purulent respiratory problems, such as bronchitis, asthma, and chronic obstructive pulmonary disease (COPD). There is a long history of folk usage with a good safety record. More recently, the biochemical details behind these effects have been clarified. Although other plant oils may be more microbiologically active, the safety of moderate doses of EO and its broad-spectrum antimicrobial action make it an attractive alternative to pharmaceuticals. EO has also been shown to offset the myelotoxicity of one chemotherapy agent. Whether this is a general attribute that does not decrease the benefit of chemotherapy remains to be determined. This article also provides instruction on how to assemble inexpensive devices for vapor inhalation.
Self-Constructed Pocket Inhaler
A suitable-sized piece of stockinet or loosely woven fabric can be rolled on a small wooden skewer and twisted into a two-dram glass vial (Patient Handout 2). The fabric easily accepts 70 drops of EO without saturation. Carrying the vial inhaler in a pocket close to the body furnishes heat for satisfactory vaporization. Hold the vial close to or touching the lips or nostrils during inhalation. The amount of vapor intake is regulated by the distance from the vial and the speed of breathing – the closer and slower, the more vapor. Best results seem to be obtained by alternating mouth and nasal inhalation during sessions of 3-5 minutes several times daily
[F1]This is a Joke—asking the wolf to watch the henhouse —there will be no protection at all here on this issue and nothing will be resolved—The FDA has constantly refused to obey congress or any other gov’t in the act of prevention ofr removal of there influence and usually has become more defiant in there quest to control the very fabric of life —and the USDA does absolutely nothing these days to enforce safety in fods and food imports nor do they do anything in regard to Chemtrails or Flouridation of water
[F2]9MG’S/1KG (2.2 LB)
TAKE YOUR weight and divide by 2.2 to get a Kg weight—example 170/2.2= 77kgs approx—then multiply by 9= 693 mgs or 3 mgs /KG— so take as a example 170/2.2===77 X 3 =231mgs