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Researchers Conclude Wireless Radiation Causes Cancer After Latest Scientific Findings Announced
National advocacy group calls on major children’s health organizations to promote safe technology in schools with the “Turn It Off 4 Kids” Initiative—PRLog Feb. 3, 2015 – The National Association for Children and Safe Technology (NACST) is taking action after two recently published studies indicate there is sufficient evidence demonstrating exposure to wireless radiation, also known as RF-EMF, causes cancer. Wireless routers and devices such as tablets, laptops, baby monitors and cell phones all emit this type of radiation.-NACST is calling on children’s health and cancer prevention organizations to make the issue of children’s health and exposure to wireless radiation in educational settings an immediate priority for 2015.
The State of the Science: The Debate is Over
Professor of Oncology Lennart Hardell, MD, PhD, and Statistician Michael Carlberg of Orebro University Hospital, Sweden found a 3-fold risk with 25 or more years of cell and cordless phone use in a study published October 2014 in Pathophysiology. Very significant was the finding that people who first used mobile or cordless phones before the age of 20 had the highest risk.-Increased wireless phone use also correlated with lower survival rates for people diagnosed with the most malignant gliomas in a second study published in the International Journal of Environmental Research and Public Health by the same researchers. Hardell and Carlberg stated, “Due to the relationship with survival, the classification is strengthened.” In both studies, the authors state that RF-EMF should be regarded as a human carcinogen, “requiring urgent revision of current exposure guidelines.”–These two studies followed the July 2014 Occupational and Environmental Medicine Journal publication of the CERENAT case controlled study where French researchers found almost a 3-fold increase in brain cancer with 896 or more hours of lifetime cell phone use.–Based on the accumulation of research demonstrating the health effects from wireless radiation, Professor Olle Johansson of the Karolinska Department of Neuroscience has stated, “the debate is over” on wireless.–
“Given the established and emerging science, it only follows that students be provided a safe learning environment, free from wireless radiation,” stated an NACST spokesperson.
Scientists Call for the World Health Organization to Reclassify RF-EMF
In 2011, the WHO’s International Agency for Research on Cancer (IARC) classified RF radiation as a Class 2B, “possible human carcinogen.” Since 2011, several of the World Health Organization invited scientists have called for a reclassification to an increased risk level. The abstracts of these 2014 studies state that RF should now be regarded as a “Group 1 Human Carcinogen,”[F1] placing it in the same category as tobacco, asbestos and benzene.
NACST’s Turn It Off 4 Kids Initiative
In light of these recent scientific publications and expert warnings, NACST is reaching out to health organizations asking them to prioritize the issue of children’s health and wireless exposures in educational settings for 2015 in the following ways:
1. Call for all new school technology to be hardwired.
2. Call to replace existing wireless technology systems with hardwired systems.
3. Call for the implementation of primary prevention efforts such as educating the public about simple steps to reduce exposure, especially in regard to children and pregnant women.
4. Educate their organization’s members and audience on this issue by emails, informational web pages, updated materials, and all other means possible.
Expert Endorsements
NASCT’s Initiative has been endorsed by several prominent scientists, physicians and safety advocates including Drs. Lennart Hardell, Olle Johansson, Anthony Miller and Dariusz Leszczynski. Dr. Leszczynski was a participating scientist in the WHO IARC panel on RF-EMF and cancer, and Dr. Miller has served as Director of the Epidemiology Unit, National Cancer Institute of Canada, Toronto.–Details on NACST’s Turn It Off 4 Kids Initiative, including endorsements, are here: http://www.nacst.org/nacst-turn-it-off-4-kids.html
About NACST
The National Association for Children and Safe Technology is dedicated to raising awareness about the health impacts of wireless radiation on children as well as advancing policies that safeguard children’s health and well being.
http://www.NACST.org
http://www.prlog.org/12421346-researchers-conclude-wireless-radiation-causes-cancer-after-latest-scientific-findings-announced.html
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Evaluation of the antibacterial potential of Petroselinum crispum-Parsley and Rosmarinus officinalis[F2] against bacteria that cause urinary tract infections.
Braz J Microbiol. 2013;44(3):829-34
Authors: Petrolini FV, Lucarini R, de Souza MG, Pires RH, Cunha WR, Martins CH
Abstract
In this study we evaluated the antibacterial activity of the crude hydroalcoholic extracts, fractions, and compounds of two plant species, namely Rosmarinus officinalis and Petroselinum crispum, against the bacteria that cause urinary tract infection. The microdilution method was used for determination of the minimum inhibitory concentration (MIC) and minimum bactericidal concentration (MBC). The crude hydroalcoholic extract of R. officinalis displayed in vitro activity against Gram-positive bacteria, with satisfactory MBC for the clinical isolate S. saprophyticus. The fractions and the pure compound rosmarinic acid did not furnish promising results for Gram-negative bacteria, whereas fractions 2, 3, and 4 gave encouraging results for Gram-positive bacteria and acted as bactericide against S. epidermidis as well as E. faecalis (ATCC 29212) and its clinical isolate. R. officinalis led to promising results in the case of Gram-positive bacteria, resulting in a considerable interest in the development of reliable alternatives for the treatment of urinary infections. -PMID: 24516424 [PubMed – indexed for MEDLINE]
Recipe for this take equal parts of each and boil as a tea or make an extract or tincture by using the alcohol to pull out the components—you can use either the blender method or the soaking for this
When done would use teaspoon increments 1 tsp every 3 hours
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Chinese Hawthorn Berry
Effects of vitexin-2″-O-rhamnoside and vitexin-4″-O-glucoside on growth and oxidative stress-induced cell apoptosis of human adipose-derived stem cells.
J Pharm Pharmacol. 2014 Jul;66(7):988-97
Authors: Wei W, Ying X, Zhang W, Chen Y, Leng A, Jiang C, Liu J
Abstract
OBJECTIVES: Vitexin-2″-O-rhamnoside (VOR) and vitexin-4″-O-glucoside (VOG) are the two main flavonoid glycosides of the leaves of Cratagus pinnatifida Bge. var. major N. E. Br.[F3] that has been widely used for the treatment of cardiovascular system diseases. In this study, we simultaneously investigated the influence of VOR and VOG on human adipose-derived stem cells (hADSCs) injury induced by hydrogen peroxide (H2 O2 ) to further characterize their anti-oxidative and anti-apoptotic activity. METHODS: hADSCs were isolated, cultured in vitro and pretreated with 62.5 μm VOR or 120 μm VOG for 24 h and then exposed to 500 μm H2 O2 for an additional 4 h. KEY FINDINGS: Pretreatment of hADSCs with VOR and VOG was demonstrated to significantly ameliorate the toxicity and apoptosis effects, such as morphological distortion, nuclear condensation, decreased intracellular caspase-3 activity and percentage of cells in apoptosis/necrosis by using morphological assay, immunocytochemistry and flow cytometric evaluation[F4]. In addition, VOR and VOG caused no cytotoxic effect on hADSCs at concentrations up to 250 and 480 μm, respectively.–CONCLUSIONS: Our results indicated that both VOR and VOG contribute to the protection against H2 O2 -mediated oxidative stress damage and could be safely used for a wide range of concentrations.[F5] PMID: 24533889 [PubMed – indexed for MEDLINE]
Recipe—Percolate the leaves and or fruits of hawthorn berry—make jams—preserves—fuse in oil—and access it as needed or take daily as a tea tincture or fused with a good oil
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New study postulates the role of dietary advanced glycation end products in the risk of Alzheimer’s disease
Date:
February 3, 2015
Source:
IOS Press BV
A new paper published in the Journal of Alzheimer’s Disease provides evidence that cooking foods at high temperatures increases the risk of Alzheimer’s disease. This study looked at the content of advanced glycation end products (AGEs) in national diets and clinical studies comparing and compared total AGEs to Alzheimer’s disease rates.—AGEs are a group of compounds that are combinations of sugars and proteins and other large molecules.[F6] They can be formed in the body, and there is a large body of literature on AGEs and Alzheimer’s disease. However, AGEs are also formed when food is cooked at high temperatures or aged for a long time such as in hard cheese. AGEs increase the risk of various chronic diseases through several mechanisms including increased inflammation and oxidative stress. They can also bind to the receptor for AGEs (RAGE). RAGE transports beta-amyloid proteins across the blood-brain barrier and contributes to the development of Alzheimer’s disease.–Our newly published paper is the first that estimated the AGE content of diets from observational studies in various countries, which estimated the link between dietary factors and risk of Alzheimer’s disease.[F7] For this purpose, the values for AGE for many types of food were taken from a study by researchers at the Mount Sinai School of Medicine in New York. They cooked 549 foods by different methods and measured the AGE content of the cooked food..–To use their findings in our study They found that the higher the cooking temperature, the higher the AGE content. For example, 100 grams of raw beef had 707 kU of AGEs, but 100 grams of roast beef had 6071 kU, we obtained information from observational studies in which diet was assessed using food frequency questionnaires or from national dietary supply values from the Food and Agriculture Organization of the United Nations. We then used either a range of cooking temperatures or methods for the observational studies or an estimate of average cooking methods and temperatures for the national dietary supply data.–In typical national diets, we found that meat made the highest contribution of AGEs, followed by vegetable oils, cheese, and fish. Foods such as cereals/grains, eggs, fruit, legumes, milk, nuts, starchy roots, and vegetables generally make low contributions to the total amount of AGEs in a diet, either because they are generally prepared at low temperatures or since they comprise smaller portions of diet[F8]s.–According to Drs. Jaime Uribarri and Weijing Cai of The Icahn School of Medicine at Mount Sinai, “This epidemiological study supports our previous findings in animals and humans of an important role for dietary AGEs in Alzheimer’s disease. We found that mice kept on a diet high in AGEs, similar to Western diet, had high levels of AGEs in their brains together with deposits of amyloid-β, a component of the plaques characteristic of Alzheimer’s disease, while at the same time developed declines in cognitive and motor abilities. The mice fed a low AGE diet remained free of these conditions. In addition, clinical studies have shown that subjects with higher blood AGE levels, in turn resulting from high AGE diets, are more likely to develop cognitive decline on follow up.–[F9]The findings point to an easily achievable goal that could reduce the risk of dementia through the consumption of non-AGE-rich foods, for example, foods that cooked or processed under lower heat levels and in the presence of more water, raising the importance of not just what we eat, but also how we prepare what we eat.[F10]”–Story Source-The above story is based on materials provided by IOS Press BV. Note: Materials may be edited for content and length.–Journal Reference–Perrone L, Grant WB. Observational and ecological studies of dietary advanced glycation end products in national diets and Alzheimer’s disease incidence and prevalence. Journal of Alzheimer’s Disease, February 2015 DOI: 10.3233/JAD-140720
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Damaged DNA amplified by activities such as smoking
Date:
January 15, 2015
Source:
ETH Zurich
In the majority of cases, the onset of cancer is characterised by a minor change in a person’s genetic material. A cell’s DNA mutates in a particular area to the extent that the cell no longer divides in a controlled manner, but begins to grow uncontrollably. In many cases, this type of genetic mutation involves chemical changes to individual building blocks of DNA. These changes are induced by smoking tobacco and consuming foods such as cured meats[F11]. This is because the contents of these materials can chemically react with and change building blocks of cellular DNA, thereby creating DNA adducts. Up to now, scientists have been able to determine whether gene samples contain adducts and if so, how many. However, the procedure is laborious and finding out exactly where a building block in the genetic code has been altered into an adduct has not been possible.—Researchers from the team led by Shana Sturla, professor of Food and Nutrition Toxicology, have succeeded for the first time in amplifying gene samples containing DNA adducts while retaining references to these adducts. This type of amplification is a prerequisite for the majority of technologies used by researchers to determine a gene’s DNA sequence. In the future, it may therefore be possible to expand DNA sequencing from the four basic DNA building blocks to include adducts. “The scientific community would have an important tool for making a detailed analysis of the molecular mechanisms involved in the initiation of cancer and the corresponding risk factors,” says Sturla.—Artificial counterpart found—The researchers focused their efforts on a specific, typical DNA adduct, an alkylguanine called O-6-benzylguanine. They recreated an enzyme reaction in a test tube to obtain a negative copy of the genetic material — analogous to how DNA is replicated naturally in cells. The scientists first had to find an artificial counterpart of the alkylguanine to be incorporated into the negative copy in its position — due to the fact that nature produces molecular counterparts to the basic DNA building blocks, but not to DNA adducts. This is why replicating genes usually leads to copy errors (or mutations) when adducts are present.–The ETH researchers produced several artificial derivatives of the basic DNA building blocks in the laboratory and tested them as potential counterparts to the alkylguanine. One proved particularly suitable. The researchers were then able to produce a negative copy of a gene containing the alkylguanine.–The aim of the work carried out by Sturla and her colleagues was to demonstrate that it is feasible to amplify genes even when adducts are present. It should now be possible for researchers to find artificial counterparts to other adducts using the same method. As the ETH Professor points out, this means that altered genes could be amplified in the future and their sequences more easily ascertained. In 2010, Shana Sturla was awarded a five-year ERC Starting Grant from the European Research Council. The current project was partly financed by this award.-Story Source-The above story is based on materials provided by ETH Zurich. The original article was written by Fabio Bergamin. Note: Materials may be edited for content and length.-Journal Reference-Laura A. Wyss, Arman Nilforoushan, Fritz Eichenseher, Ursina Suter, Nina Blatter, Andreas Marx, Shana J. Sturla. Specific Incorporation of an Artificial Nucleotide Opposite a Mutagenic DNA Adduct by a DNA Polymerase. Journal of the American Chemical Society, 2015; 137 (1): 30 DOI: 10.1021/ja5100542
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Aging impacts epigenome in human skeletal muscle
Date:
November 20, 2013
Source:
Buck Institute for Research on Aging
Our epigenome is a set of chemical switches that turn parts of our genome off and on at strategic times and locations. These switches help alter the way our cells act and are impacted by environmental factors including diet, exercise and stress[F12]. Research at the Buck Institute reveals that aging also effects the epigenome in human skeletal muscle. The study, appearing on line in Aging Cell, provides a method to study sarcopenia, the degenerative loss of muscle mass that begins in middle age.—The results came from the first genome-wide DNA methylation study in disease-free individuals. DNA methylation involves the addition of a methyl group to the DNA and is involved in a particular layer of epigenetic regulation and genome maintenance. In this study researchers compared DNA methylation in samples of skeletal muscle taken from healthy young (18 — 27 years of age) and older (68 — 89 years of age) males. Buck faculty and lead scientist Simon Melov, PhD, said researchers looked at more than 480,000 sites throughout the genome. “We identified a suite of epigenetic markers that completely separated the younger from the older individuals — there was a change in the epigenetic fingerprint,” said Melov. “Our findings were statistically significant; the chances of that happening are infinitesimal.”–Melov said scientists identified about six-thousand sites throughout the genome that were differentially methylated with age and that some of those sites are associated with genes that regulate activity at the neuromuscular junction which connects the nervous system to our muscles. “It’s long been suspected that atrophy at this junction is a weak link in sarcopenia, the loss of muscle mass we get with age,” said Melov. “Maybe this differential methylation causes it. We don’t know.”–Studying the root causes and development of sarcopenia in humans is problematic; the research would require repeated muscle biopsies taken over time, something that would be hard to collect.[F13] Melov says now that the epigenetic markers have been identified in humans, the goal would be to manipulate those sites in laboratory animals. “We would be able to observe function over time and potentially use drugs to alter the rate of DNA methylation at those sites,” he said. Melov says changes in DNA methylation are very common in cancer and that the process is more tightly controlled in younger people.-Story Source-The above story is based on materials provided by Buck Institute for Research on Aging. Note: Materials may be edited for content and length.-Journal Reference-Simon Melov, PhD et al. Genome-wide DNA methylation changes with age in disease-free human skeletal muscle. Aging Cell, November 2013
TOP
[F1]This is huge—the implication here should lead to huge lawsuits and force the telecommunications industry to come up with better shielding of this radiation leaking into the brain
[F2]Parsely and Rosemary—would either water extract as a tea combo or alcohol extract for tincture
[F3]Hawthorn Berry
[F4]Plain English it stopped death
[F5]Strong as you like
[F6]This causes glycation when the sugars and proteins bind together—causing huge amounts of free radicals so when cooking proteins with carbs can cause this to happen as well
[F7]Another factor that is not considered when adding glyphosates into the foods and genetics with the sugars and proteins this to causes a chemical reaction and when adding nano silver —the sugar makes the silver more toxic to the body—-
[F8]These are not safer in the sense that they are relatively free—these are less toxic due to less consumption—but to offset the effect of AGE—things like B1 and MSM both have anti glycating effects
[F9]Again this is when High heat and proteins and carbs( sugars) are cooked together —always remember the concept of glycation protein + sugar connected chemically when heated at high heat causes damage
[F10]This is Key
If your cooking or fusing proteins with sugars this is what will cause the issues—if your diet is relatively high in sugar then you can as well cause this effect
[F11]This is an old report —we know today that almost anything being consumed with genetics—glyphosates –endocrine disrupting chemicals—nanoparticles—metals –phytoestrogenic foods will also cause this kind of mutation
[F12]Or environmental pollutants –Genetics—PhytoCheistry—Pesticides—Metal exposure
[F13]Unless you were harvesting people from hospitals
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TOPA
HOME
Show of the Month February 14 2015
Pharma Lies To Doctors about The Medicine You are Taking
Marketable Window Dressing
Vitamin A for treating measles in children
Vitamin A may Counteract these Potentially Toxic Substances
Vitamin A supplementation for preventing morbidity and mortality in children from 6 months to 5 years of age
Vitamin A’s Immune System Health Benefits
How much sleep do we really need?
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Pharma Lies To Doctors about The Medicine You are Taking
By: Shane Ellison, MS
Following doctor’s orders has become synonymous with danger.
In my book, Over-The-Counter Natural Cures, I documented that every year, FDA- approved drugs kill twice as many people as the total number of U.S. deaths from the Vietnam War.
Death by medicine flourishes because deceit, not science, governs a doctor’s prescribing habits. –Working as a pharmaceutical chemist, I learned that the deceit comes in many forms. –Medical ghostwriting and checkbook ‘science’ are the most prominent. –Doctors rely on peer-reviewed medical journals to learn about prescription drugs. –These journals include the Lancet, British Medical Journal, New England Journal of Medicine and the Journal of the American Medical Association. –It’s assumed that these professional journals offer the hard science behind any given drug. This assumption is wrong. –Thanks to medical ghost-writing, medical journals can’t be trusted. –Medical ghostwriting is the practice of hiring Ph.D.s to crank out drug reports that hype benefits while hiding negative side effects. –Once complete, drug companies recruit doctors to put their name on the report as the authors.–These reports are then published in the above mentioned medical journals. –The carrot for this deceitful practice is money and prestige. Ghostwriters can receive up to $20,000 per report. Doctors receive prestige from having been published.–As deplorable as medical ghostwriting sounds, it is more common than you think. –Dr. Jeffrey Drazen, editor for the New England Journal of Medicine, insists that he cannot find drug review authors who do not have financial ties to drug companies. –Dr. David Healy, of the University of Wales, predicts that 50% of the journals drug review articles are written by ghostwriters hired by Big Pharma.–The editor of the British Journal of Medicine has acknowledged that medical ghostwriting has become a serious problem for his publication: “We are being hoodwinked by the drug companies. –The articles come in with doctors’ names on them and we often find some of them have little or no idea about what they have written.”–Consider the testimony from deputy editor of the Journal of the American Medical Association: –“This [journal articles] is all about bypassing science. –Medicine is becoming a sort of Cloud Cuckoo Land, where doctors don’t know what papers they can trust in the journals, and the public doesn’t want to believe.”
Confessions of Ghostwriters—-
Ex-medical ghostwriter Susanna Rees stated: –“Medical writing agencies go to great lengths to disguise the fact that the papers they ghostwrite and submit to journals and conferences are ghostwritten on behalf of pharmaceutical companies and not by the named authors,’ she wrote. ‘There is a relatively high success rate for ghostwritten submissions ─ not outstanding, but consistent.”–Other ghostwriters have come forward privately:
Ghostwriter 1–“I agreed to do two reviews for a supplement to appear under the names of respected ‘authors.’ I was given an outline, references, and a list of drug-company approved phrases.-I was asked to sign an agreement stating that I would not disclose anything about the project. I was pressured to rework my drafts to position the product more favorably.”
Ghostwriter 2–“I was told exactly what the drug company expected and given explicit instructions about what to play up and what to play down.”
NSAID Popularity Courtesy of Ghostwriting not Science
To illustrate the negative impact of medical ghostwriting, we can look to commonly used non-steroidal anti-inflammatory drugs (NSAIDs). –Between 1990 and 1997, all clinical trials performed on NSAIDS such as Vioxx, Aleve, Aspirin, Motrin, and Ibuprofen, were sponsored by the drug manufacturers. –The result was that 100% of the studies showed the sponsored drug to have equal or superior efficacy when compared to other drugs. –Thus, according to studies done from 1990-1997, every NSAID drug tested during this time was superior to every other NSAID product… all at the same time! –The fallacies behind medical ghost writing on NSAIDS are exposed through injuries and deaths among users.–Approximately 107,000 patients are hospitalized every year for NSAID-related gastrointestinal complications. –Vioxx alone injured 100,000 during its rein as king of pain killers. –The risk of miscarriage for women who take the NSAID aspirin is 60 percent higher than for those who do not. –At least 16,500 NSAID-related deaths occur each year among arthritis patients. This figure is comparable to the number of deaths from the so-called acquired immunodeficiency syndrome (AIDS). In fact, NSAIDS contribute to as many deaths as multiple myeloma, asthma, and cervical cancer combined. –These statistics do not account for over-the-counter use of NSAIDS, only for arthritis patients. –We can be confident that there are considerably more deaths caused by the use of NSAIDS that go unreported. –And because few medical doctors are unaware of these statistics, NSAIDS can rightfully be considered a silent killer. –This is especially true when “experts” are paid by Big Pharma to write favorable reviews while hiding dangerous side effects.
Buying Results, Professors and Government
Other weapons of mass deception exist ─ ‘checkbook science.’ –As defined by Diana Zuckerman, Ph.D., checkbook science is research intended not to expand knowledge or to benefit humanity but instead to sell products [drugs].–It has stolen the very soul of University research, scientific method, and the patients who serve as human subjects.–Checkbook science explains why deadly drugs are approved. –Leveraging their financial power, drug companies structure the protocol designed to study whether or not a drug is safe. They choose the investigators (from academics and government institutions) and in many instances are involved in the collation, interpretation and reporting of data. Akin to medical ghostwriting, this practice allows drug companies to hide the dangers associated with drugs while highlighting benefits.5 As with medical ghostwriting, checkbook science is more common than you think. A third of academic professors have personal financial ties to drug makers. Government institutions are guilty, too. Called the “Stealth Merger” by The LA Times, top scientists at the National Institutes of Health also collect paychecks and stock options from the drug industry[F1].-Once considered “an island of objective and pristine research, untainted by the influences of commercialization,” the National Institutes of Health has become corrupted by checkbook science. -To substantiate, we look to the following statistics from the LA Times:–Dr. Stephen I. Katz, director of the NIH’s National Institute of Arthritis and Musculoskeletal and Skin Diseases collected between $476,369 and $616, 365 in fees over a ten-year period.——-From 1997-2002, Dr. John I. Gallin, director of the NIH’s Clinical Center, received between $145,000 and $322,000 in fees and stock proceeds from the drug industry.—Dr. Richard C. Eastman is the NIH’s top diabetes researcher.-As a consultant to the drug manufacturers in 1997, he wrote to the Food and Drug Administration (FDA) defending a product without disclosing his conflict of interest. –His letter stated that the risk of liver failure from the given drug was “very minimal.” —Six months later, a patient taking the drug in an NIH study that Eastman oversaw, Audrey LaRue Jones, suffered sudden liver failure and died–An autopsy, along with liver experts, found that the drug had caused the liver failure.—Dr. Ronald N. Germain, deputy director of a major laboratory at the National Institute of Allergy and Infectious Diseases, amassed more than $1.4 million od Big Pharma money in “consulting fees” from 1993 to 2003, plus stock options.
Jeffrey Schlom, director of the National Cancer Institute’s Laboratory of Tumor Immunology and Biology, received $331,500 in company fees over 10 years.—Jeffrey M. Trent, who became scientific director of the National Human Genome Research Institute in 1993, reported between $50,608 and $163,000 in industry consulting fees. He left the government in 2002.
Lying to Doctors is Legal
Checkbook science has been going on for more than 20 years. Known as the Bayh-Dole Act, U.S patent law was amended in 1980 to allow for these flagrant conflicts of interest. –Heart drugs introduced in the 1970‘s serve as an excellent example of how checkbook science damages the public’s health. —By 1990, they were estimated to kill more Americans than the 58,000 killed in the Vietnam War.–This disaster could have been avoided. –Early research suggesting that these drugs were lethal would have saved thousands of lives. –Putting checkbook science to work, these risky research findings went unpublished by the pharmaceutical company that funded the research.–Children suffer, too, from checkbook science. Checkbooks science was responsible for motivating doctors to push antidepressants on this vulnerable population.[F2]
Checkbook Science Gets Antidepressant Approved for Kids!
Published research paid for by drug manufactures showed antidepressant drugs (selective serotonin reuptake inhibitors) to be safe and effective for children. –These drugs include Paxil and Prozac. -Conversely, when unpublished results were finally obtained it was discovered that depressed children taking antidepressants were twice as likely to become suicidal as children taking a placebo. –Acknowledging the deceit, the Lancet stated: “The story of research into SSRI use in childhood depression is one of confusion, manipulation, and institutional failure.”7
Drug Advertising Gets You Hooked
Hopefully the line at the pharmaceutical trough will grow shorter as the medical ghostwriting and checkbook science scandal becomes public. –Yet drug makers have an insurance policy for this ─ Direct-to-Consumer advertising. The oft repeated “ask your doctor” ensures that the herd instinctively embraces drugs, drugs, and more drugs.
Doctors Only See Positive, Not Negative, You Suffer
Understanding medical ghost-writing and checkbook science explains why medical doctors have been hypnotized into drug worship ─ they only see the positive[F3]. –It also explains why modern medicine is more deadly and lucrative than war ─ the danger has been silenced with the pen and money. -In sum, these methods of deceit ensure that doctors are not keen to the dangers of prescription drugs. –Drug companies do not take responsibility for the wanton prescription drug deceit. –Instead, victims have been made invisible – dehumanized. –They are not recognized as children or as men with a significant contribution to society.–Instead their deaths are attributed to them being sick or just too damn old[F4]. —Those who profit from prescription drugs should hold some sort of record for demonstrating the most reckless disregard for human life.–If the deceit continues the prescription drug leviathan will silently kill more people than Napalm dropped on Vietnamese villages. Realize –research and make your own remedies—utilizing things that will not cause more problems then you already are dealing with
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Marketable Window Dressing
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Vitamin A for treating measles in children
Huiming Y1, Chaomin W, Meng M.
Author information
Abstract
BACKGROUND:
Measles is a major cause of childhood morbidity and mortality. Vitamin A deficiency is a recognized risk factor for severe measles infections. The World Health Organization (WHO) recommends administration of an oral dose of vitamin A (200,000 international units (IU), or 100,000 IU in infants) each day for two days to children with measles when they live in areas where vitamin A deficiency may be present.
OBJECTIVES:
To determine whether vitamin A therapy, commenced after measles has been diagnosed, is beneficial in preventing mortality, pneumonia and other secondary complications in children.
SEARCH STRATEGY:
We searched the Cochrane Central Register of Controlled Trials (CENTRAL) (The Cochrane Library Issue 1, 2005), MEDLINE (1966 to March 2005), EMBASE (1980 to December 2004) and looked for unpublished studies.
SELECTION CRITERIA:
Only randomized controlled trials in which children with measles were given vitamin A or placebo along with standard treatment were considered.
DATA COLLECTION AND ANALYSIS:
Studies were assessed independently by two authors. The analysis of dichotomous outcomes was done using the StatXact software and results expressed as relative risk (RR) with 95% confidence interval (CI). Subgroup analyses were carried out for dose, formulation, age, hospitalization and pneumonia-specific mortality. Weighted mean differences (WMD) with 95% CI were calculated for continuous outcomes.
MAIN RESULTS:
There was no significant reduction in the risk of mortality in the vitamin A group when all the studies were pooled using the random-effects model (RR 0.70; 95% CI 0.42 to 1.15). Using two doses of vitamin A (200,000 IU) on consecutive days was associated with a reduction in the risk of mortality in children under the age of two years (RR 0.18; 95% CI 0.03 to 0.61) and a reduction in the risk of pneumonia-specific mortality (RR 0.33; 95% CI 0.08 to 0.92). There was no evidence that vitamin A in a single dose was associated with a reduced risk of mortality among children with measles. There was a reduction in the incidence of croup (RR 0.53; 95% CI 0.29 to 0.89) but no significant reduction in the incidence of pneumonia (RR 0.92; 95% CI 0.69 to 1.22) or diarrhoea (RR 0.80; 95% CI 0.27 to 2.34) with two doses.
AUTHORS’ CONCLUSIONS:
Although we found no overall significant reduction in mortality with vitamin A therapy for children with measles there was evidence that two doses were associated with a reduced risk of mortality and pneumonia-specific mortality in children under the age of two years. There were no trials that directly compared a single dose with two doses.
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Vitamin A may Counteract these Potentially Toxic Substances
Electromagnetic Radiation
Vitamin A (large dosages of 35,000 – 100,000 IU per day) may improve the general condition of persons undergoing Radiation Therapy
: references
Vitamin A may help to prevent Radiation Therapy-induced Pneumonitis (if Vitamin A therapy is commenced prior to Radiation Therapy).
Vitamin A (especially the Retinyl Palmitate form of Vitamin A applied topically) may inhibit the ability of UV-B to cause Sunburn (i.e. topically applied Retinyl Palmitate may function as a Sunscreen). references
Environmental Toxins
Vitamin A may help to protect the body from the toxic effects of Air Pollution. references
Vitamin A may counteract some of the toxic effects of Dioxin exposure. references
Enzymes
Vitamin A (Retinol and Retinoic Acid forms) may inhibit Lipoxygenase. references
Vitamin A may inhibit Ornithine Decarboxylase. references
Hormones
Vitamin A may help to lower elevated Cortisol levels. references
Immune System Chemicals
Vitamin A may inhibit the production of excessive quantities of Tumor Necrosis Factor (TNF). references
Minerals
Vitamin A may facilitate the detoxification of Lead from the body. [more info]
Pharmaceutical Drugs
Vitamin A (administered concurrently with Methotrexate) may inhibit the damage to the Small Intestine caused by Methotrexate. references
Vitamin A (Retinol) may counteract and prevent the suppression of the Immune System that is caused by Pharmaceutical Glucocorticosteroids. references
Proteins
Vitamin A may counteract the toxicity of Amyloid-Beta Protein. references
Vitamin A may lower elevated Fibrinogen levels. references
Vitamin A may inhibit the activation of Nuclear Factor-Kappa B (NF-Kappa B). references
Recreational Drugs
Vitamin A may counteract the toxic effects of Tobacco smoking (by strengthening the Mucous Membranes of the Bronchial Tubes and Lungs): references
Vitamin A may inhibit the ability of Tobacco to cause Emphysema.
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Vitamin A supplementation for preventing morbidity and mortality in children from 6 months to 5 years of age.
Imdad A1, Herzer K, Mayo-Wilson E, Yakoob MY, Bhutta ZA.
Author information
Abstract
BACKGROUND:
Vitamin A deficiency (VAD) is a major public health problem in low and middle income countries affecting 190 million children under 5. VAD can lead to many adverse health consequences, including death.
OBJECTIVES:
To evaluate the effect of vitamin A supplementation (VAS) for preventing morbidity and mortality in children aged 6 months to 5 years.
SEARCH STRATEGY:
We searched the Cochrane Central Register of Controlled Trials (CENTRAL 2010 Issue 2), MEDLINE (1950 to April Week 2 2010), EMBASE (1980 to 2010 Week 16), Global Health (1973 to March 2010), Latin American and Caribbean Health Sciences (LILACS), metaRegister of Controlled Trials and African Index Medicus (27 April 2010).
SELECTION CRITERIA:
Randomised controlled trials (RCTs) and cluster RCTs evaluating the effect of synthetic VAS in children aged 6 months to 5 years living in the community. We excluded studies concerned with children in hospital and children with disease or infection. We excluded studies evaluating the effects of food fortification, consumption of vitamin A rich foods or beta-carotene supplementation.
DATA COLLECTION AND ANALYSIS:
Two review authors independently assessed studies for inclusion. Data were double abstracted and discrepancies were resolved by discussion. Meta-analyses were performed for outcomes including all-cause and cause-specific mortality, disease, vision, and side-effects.
MAIN RESULTS:
43 trials involving 215,633 children were included. A meta-analysis for all-cause mortality included 17 trials comprising 194,795 children with 3536 deaths in both groups. At follow-up, there was a 24% observed reduction in the risk of all-cause mortality for Vitamin A compared with Control (Relative risk (RR) = 0.76 [95% confidence interval (CI) 0.69, 0.83]). Seven trials reported diarrhoea mortality and a 28% overall reduction for VAS (RR = 0.72 [0.57, 0.91]). There was no significant effect of VAS on cause specific mortality of measles, respiratory disease and meningitis. VAS reduced incidence of diarrhoea (RR = 0.85 [0.82, 0.87]) and measles morbidity (RR = 0.50 [0.37, 0.67]); however, there was no significant effect on incidence of respiratory disease or hospitalisations due to diarrhoea or pneumonia. There was an increased risk of vomiting within the first 48 hours of VAS (RR = 2.75 [1.81, 4.19]).
AUTHORS’ CONCLUSIONS:
VAS is effective in reducing all-cause mortality by about 24% compared to no treatment. In our opinion, given the evidence that VAS causes considerable reduction in child mortality, further placebo-controlled trials of VAS in children between 6 months and 5 years of age are not required. There is a need for further studies comparing different doses and delivery mechanisms (for example, fortification).
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Vitamin A’s Immune System Health Benefits
Immune System: Ailments
Vitamin A may help to prevent most Bacterial & Viral Diseases and Vitamin A deficiency increases susceptibility to Bacterial & Viral Diseases (via numerous mechanisms that involve the Immune System): references
Vitamin A may help to prevent infections from Viruses: references
Vitamin A may be useful for the treatment of Acquired Immune Deficiency Syndrome (AIDS): references
Vitamin A may retard the onset of full-blown AIDS in persons who are infected with the HIV virus.
High Vitamin A concentrations may suppress the replication of the HIV virus in Macrophages.
Vitamin A deficiency has been correlated with increased (earlier) mortality in AIDS patients.
Vitamin A may help to increase the number of circulating Helper T-Cells in AIDS patients.
Vitamin A supplementation (during early Pregnancy) dramatically reduces the rate of vertical transmission (i.e. from mother to infant) of the HIV virus.
Vitamin A deficiency may increase the body’s susceptibility to Chickenpox infection. references
Vitamin A (50,000 – 150,000 IU per day for three to five days) may exert anti-viral effects against the Viruses that cause the Common Cold. references
Vitamin A (50,000 – 150,000 IU per day for three to five days) may exert anti-viral effects against the Viruses that cause Influenza. references
Vitamin A may deactivate the Herpes Simplex Virus Type 2. references
Vitamin A reduces the mortality rate in children infected with Measles by up to 50%. references
Vitamin A deficiency may increase the risk of (meningococcal) Meningitis. references
Vitamin A (12,500 – 25,000 IU per day) significantly reduces the severity of the Respiratory Syncytial Virus (RSV). references
Vitamin A deficiency may exacerbate the severity of Rotavirus infection. references
Vitamin A may accelerate the recovery from Shigella infections (but does not inhibit or kill Shigella species). references
Vitamin A may reduce Inflammation. references
Vitamin A (100,000 IU daily for two weeks) may improve various impairments of the function of the Immune System in Systemic Lupus Erythematosus (SLE) patients. references
Vitamin A (Retinol form) may help to prevent Malaria (by suppressing the Plasmodium falciparum Protozoa that causes Malaria). references
Immune System: Ailments: Cancer
Vitamin A may help to prevent many types of Cancer and Vitamin A therapy may suppress the further growth of the (already established) tumors involved in some types of Cancer: references
Vitamin A may may help to prevent Basal Cell Carcinoma. references
Vitamin A (40,000 IU per day) may reduce the recurrence of Bladder Cancer tumors in people with existing Bladder Cancer by up to 53%. references
Vitamin A may help to prevent Breast Cancer. references
Vitamin A may help to prevent Cervical Cancer. references
Vitamin A may help to prevent Colon Cancer. references
Vitamin A may help to prevent Endometrial Cancer. references
Vitamin A may help to prevent Esophageal Cancer. references
Vitamin A (100,000 IU per day) “may” help to treat Glioblastoma Multiforme.
The Retinyl Palmitate (300,000 IU – 1,500,000 IU per day, caution: a high dosage) form of Vitamin A may help to prevent Larynx Cancer (laryngeal cancer). references
The Retinoic Acid form of Vitamin A (administered orally) may “direct” the cancerous cells involved in Leukemia to mature and die like normal cells.
Vitamin A may help to prevent Liver Cancer. references
Vitamin A may inhibit the tumor promotion stage of Lung Cancer. references
Vitamin A may inhibit the development of the tumors associated with Mouth Cancer. references
Vitamin A may help to prevent and treat Ovarian Cancer. references
Vitamin A may help to prevent Pharynx Cancer (Pharyngeal Cancer) by strengthening the Mucous Membranes of the Pharynx. [more info]
Vitamin A may help to prevent Prostate Cancer by strengthening the Mucous Membranes of the Prostate. references
Vitamin A may prevent the initiation and/or progression of Skin Cancers by stimulating normal Cell differentiation: references
Vitamin A may inhibit the growth and metastasis of malignant Melanoma. references
The Retinol form of Vitamin A may help to prevent Squamous Cell Carcinoma. references
Stomach Cancer references
Testicle Cancer [more info]
Vitamin A may help to prevent or regress Tongue Cancer. references
Vitamin A may help to prevent carcinogens-induced Uterus Cancer. references
Supplemental Vitamin A increases the effectiveness of conventional medical treatments for Cancer (such as Surgery, Chemotherapy and Radiation Therapy).
Immune System: Underyling Mechanisms
Vitamin A may stimulate various aspects of the Immune System: references
Vitamin A may increase the effectiveness of the Cells that produce Antibodies and Vitamin A deficiency may cause impairment in the response of Antibodies to challenges by Antigens. references
Vitamin A deficiency may cause a reduction in the production of B-Lymphocytes. references
Vitamin A deficiency may cause a decline in the production of Helper T-Cells. references
Vitamin A may increase the proliferation of Lymphocytes in response to challenges by Antigens and Mitogens. references
Vitamin A may enhance the function of Macrophages. references
Vitamin A may enhance the function of Neutrophils. references
Vitamin A deficiency impairs the function of NK Lymphocytes. references
Vitamin A deficiency causes degeneration and atrophy of the Spleen. references
Vitamin A may protect and strengthen the Thymus and supplemental Vitamin A may cause the Thymus to (beneficially) double in size. references
Vitamin A may enhance the ability of the Thymus to manufacture T-Lymphocytes and Vitamin A deficiency may cause impairment of T-Lymphocyte response. references
Vitamin A may enhance the function of White Blood Cells. references
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Vitamin A for treating measles in children.
D’Souza RM1, D’Souza R.
Author information
Update in
Cochrane Database Syst Rev. 2002;(1):CD001479.
Abstract
BACKGROUND:
Measles is a leading cause of childhood morbidity and mortality. Vitamin A deficiency is a recognised risk factor for severe measles. The World Health Organization (WHO) recommends administration of an oral dose of 200,000 IU (or 100,000 IU in infants) of vitamin A per day for two days to children with measles in areas where vitamin A deficiency may be present.
OBJECTIVES:
The purpose of this review is to determine whether vitamin A when commenced after measles has been diagnosed, is beneficial in preventing mortality, pneumonia and other complications in children.
SEARCH STRATEGY:
MEDLINE and the Cochrane Library, Issue 4, 1999 were searched.
SELECTION CRITERIA:
Only randomized controlled trials in which children with measles were given vitamin A or placebo along with standard treatment were considered.
DATA COLLECTION AND ANALYSIS:
Studies were assessed independently by two reviewers. The analysis of dichotomous outcomes was done using the StatExact software package. Sub-group analyses were done for dose, formulation, age, hospitalisation and pneumonia specific mortality. Weighted mean difference with 95% CI were calculated for continuous outcomes.
MAIN RESULTS:
The relative risks (RR) and 95% Confidence Intervals (CI) are based on the estimates from the StatExact software package. There was no significant reduction in mortality in the vitamin A group when all the studies were pooled together (RR 0.60; 95% CI 0.32 to 1.12)(Statexact estimate). There was a 64% reduction in the risk of mortality in children who were given two doses of 200,000 IU of vitamin A (RR=0.36; 95% CI 0.14 to 0.82) as compared to placebo. Two doses of water based vitamin A were associated with a 81% reduction in risk of mortality (RR=0.19; 95% CI 0.02 to 0.85) as compared to 48% seen in two doses of oil based preparation[F5] (RR=0.52; 95% CI 0.16 to 1.40). Two doses of oil and water based vitamin A were associated with a 82% reduction in the risk of mortality in children under the age of 2 years (RR=0.18; 95% CI 0.03 to 0.61) and a 67% reduction in the risk of pneumonia specific mortality (RR=0.33; 95% CI 0.08 to 0.92). There was no evidence that vitamin A in a single dose of 200,000 IU was associated with a reduced risk of mortality among children with measles (RR=0.77; 95% CI 0.34 to 1.78). Sub-groups like age, dose, formulation, hospitalisation and case fatality in the study area were highly correlated and there were not enough studies to separate out the individual effects of these factors. There was a 47% reduction in the incidence of croup (RR=0.53; 95% CI 0.29 to 0.89), while there was no significant reduction in the incidence of pneumonia (RR=0.92; 95% CI 0.69 to 1.22) or of diarrhoea (RR=0.80; 95% CI 0.27 to 2.34). Duration of diarrhoea was measured in days and there was a reduction in its duration of almost two days WMD -1.92, 95% CI -3.40 to -0.44. Only one study evaluated otitis media and found a 74% reduction in its incidence (RR=0.26, 95% CI, 0.05 to 0.92). We did not find evidence that a single dose of 200,000 IU of vitamin A per day, given in oil-based formulation in areas with low case fatality, was associated with reduced mortality among children with measles. However, there was evidence that the same dose given for two days was associated with a reduced risk of overall mortality and pneumonia specific mortality.